Pharmacology is developing rapidly, and the persistent opinion that any strong antibiotic causes much harm to the body can no longer be considered consistent. Antibiotics of the new generation, among other vital advantages, show that they can work carefully about vital human systems and effectively against pathogenic bacteria. The abstract and introduction are adequate as they are divided into relevant sections with clear and concise information about the issue. The new information for me is a tendency for harmful microorganisms to develop their immunity to treatment, even with the use of a new, modern and popular drug. Forms of medicines may also act differently: antibiotic tablets may fail in treatment, while injectable formulations will be more effective. Any antibiotic, even the safest, is recommended to be taken only as prescribed by a doctor. However, bacteria can develop resistance in some cases, which affects the body’s response, the fight against the virus, and clinical practice.
An upper respiratory study attempted to establish the role of antibiotic resistance in the pathogenesis of influenza-associated secondary bacterial infection (Zhang et al., 2020). This study shows that host response to influenza infection can indirectly influence antibiotic resistance gene expression in the respiratory tract, influencing microbial community structure and overall microbial gene expression (Zhang et al., 2020). This study is a predominantly qualitative analysis due to the small sample size. Finally, this study’s results show that recovery is worse without antibiotics, even to a certain degree of bacterial resistance. This information about upper respiratory infections is new and essential for me. In addition, this article defines the critical signs of the reaction to the virus under regulation conditions with low and medium resistance to bacteria.
Reference
Zhang, L., Forst, C. V., Gordon, A., Gussin, G., Geber, A. B., Fernandez, P. J.,… & Ghedin, E. (2020). Characterization of antibiotic resistance and host-microbiome interactions in the human upper respiratory tract during influenza infection. Microbiome, 8(1), 1-12. Web.